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Question
Sphingomyelins are important phospholipids found in the _____.a. pacemaker of the heart
b. kidney tubules
c. myelin sheath of nerves
d. human blood groups A, B and O
Answer
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Related questions
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Which hormone, mediated by the enzyme renin, is primarily responsible for active reabsorption of sodium ions in the distal and collecting tubules?
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Why are vitamins A and E said to be antagonists of vitamin K?
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Although for micronutrients increasing the amount of a protein by directly inducing gene expression is certainly a common mechanism, we have encountered other mechanisms by which vitamins can regulate proteins. Provide one example of posttranslational modification, being as specific as possible.
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The active form of vitamin A that regulates cell differentiation through gene expression is _____.
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For which vitamin is the Tolerable Upper Intake Level set because of increased tendency for bleeding?
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Which vitamin is associated with improved plasma membrane structure and glucose transport/metabolic control in people with type 2 diabetes?
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A genetic deficiency in _____ leads to a vitamin E deficiency syndrome.
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Q:
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If asked about the relationship of vitamin D to autoimmune disease, the best information for a dietetic professional to provide is:
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c. some evidence suggests a protective effect of vitamin D.
d. take a supplement containing 2000 IU vitamin D each day.
Q:
Which is the best technique to assess status of vitamin A liver stores?
a. serum retinol levels
b. the relative dose response
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a. neurological function
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Q:
Which vitamin directs cellular differentiation of epithelial cells?
a. riboflavin
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Q:
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a. lost 2 electrons
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c. gained 2 electrons
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Q:
Nutrients can be used to compensate for some gaps in our DNA. For example:a. Individuals with a change in the base sequence for the phenylalanine hydroxylase gene can correct the sequence of bases by eating a lot of phenylalanine.b. Obese individuals can lose body weight by eating fewer calories than they expend.c. Omega-3 fatty acids can up-regulate genes encoding for fat oxidation.d. Individuals with a polymorphism that decreases activity of a folate-metabolizing enzyme, methylene tetrahydrofolate reductase, may have more elevated plasma homocysteine than those with a normal enzyme while more folate intake can normalize plasma homocysteine for those with this polymorphism.